The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
you look at the @oe to so happily two homozygotes are actually more uncommon or less common than the a pony for homozygous oh you have to a bowi two copies and they also seem to be a so that also seems to be associated with a slightly increased where it's not of Alzheimer's disease but you know elevated cholesterol and cart and cardiovascular disease of course again in that same setting that we talked about earlier
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the two twos are very rare I've actually never seen a two - I've seen one okay they're reported at less than 1% of the population - I think I've seen two and they come with a risk reduction I believe of about 20%
and by the way that's the only genotype I've never seen it's much more rare than the 4-4 homozygotes but the tutu would come with about a 20 risk reduction relative risk reduction in Alzheimer's disease but paradoxically comes at this higher risk of ascvd
and as you said Peter April E2 which is bad for your lipids in the blood is actually protective against Alzheimer's