The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
I think truthfully I don't think they should change that much of what we do in other words even if you have an ape OE 3 or an ape OE - it should be a much lower risk gene I wouldn't let that information in any way shape or form distract or detract me from taking an all-hands-on-deck approach to avoiding dementia as Richard Isaacson said when we had him on the podcast if you have a brain you're at risk of Alzheimer's disease
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
just because he doesn't have a four doesn't mean he's not at some risk
in other words even if you have an apoe3 or an apoe2 which should be a much lower risk Gene I wouldn't let that information in any way shape or form distract or detract me from taking an all Hands-On deck approach to avoiding dementia
everybody with a brain is at risk
everybody with a brain is at risk
The authors estimate that without the strong underlying risks conferred by APOE ε3 and ε4, almost all Alzheimer’s disease and about half of all dementia cases would not occur.