The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
it really strengthens this idea that there may be lectins whether they're Nightshade lectins or other plant lectins these carbohydrate binding proteins in Plants transiting from our gut to our brain and then once they get to our brain it's not impossible to imagine that they could transmit to all the spinal nerves
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they would see that lectin move to the brain of these mice through the Vagas nerve and they actually had the lectin deposit in the basil ganglia which is where the dopaminergic neurons are so they see it as a model for Parkinson's
the concern is that people with Parkinson's disease the hypothesis is that humans with Parkinson's disease may have number one a genetic susceptibility number two leaky gut and number three exposure to lectin that transported through the Vegas nerve of the brain and affect the basil ganglia
things like lectins you know on my Instagram yesterday I posted about lectins and that in animal models of we see that in in worms and then write in mice and rats when we give these animals lectins they are actually taken up and transit through the vagus nerve to the brain and then in there are other population studies of people who have had these are humans who have had vagotomy never had the vagus nerve severed for some other medical reasons and in Danish in the Danish population that was studied when people have had vagotomy x' and the vagus nerve was severed they had a 40% lower incidence of Parkinson's disease suggesting these these this always have I posted yesterday was about a Parkinson's disease model that for some people this transit of these plant electives through the vagus nerve do the brain and then there differ differentially adjusting or changing dopaminergic signaling in the basal ganglia the substantia [ __ ] that can affect Parkinson's disease in these patients potentially
is it possible that lectins in our diet or something else in our diet is triggering an autoimmune reaction which is leading to degradation or damage to these striatal neurons neurons in the brain leading to parkinson's disease
that involves lectins and the fact that there is a series of studies done in c elegans a worm and in mice and rats showing that if you introduce specific lectins into these animals and you label them fluorescently you can see the lectins move from the gut through the vagus nerve in a retrograde fashion to the brain to the striatum the substantia [ __ ] of the brain in which they are associated with uh issues in the striatum in the dopamine the dopaminergic neurons of the basal ganglia in the um the parkinson affected regions of the brain in these animal models and that is a very fascinating thing to think that lectins could move retrograde from the gut to the brain become deposited there and cause neurodegeneration or misfolding of proteins so this is one compelling hypothesis for at least parkinson's parkinsonism