The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
and when you look in the brain Peter when you look at levels of amalo beta and of to they're not different from the mice without high levels of clotho that is unbelievable in other words they're brains are still riddled with the Alzheimer's toxins but they've been able to really thwart the effects of those toxins because they show normal cognition
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
that Mouse will live much longer um number one so it extends its lifespan number one number two it in it will normalize its cognition across its lifespan remarkably across its lifespan
when Masia looked at the synapses in these mice they saw he saw that that the synapses were all preserved so again like like this analogy where clao is really providing a helmet around each neuron it really allowed the synapses to be preserved but there was a whole bunch of amalo and TOA still there and that is resilience
we back crossed Alzheimer's models with clotho overexpressing models to create mice who have um mous Heimer and then have mous Heimer plus lots of clotho