The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it really shows the importance of maintaining the blood-brain barriers so that it doesn't allow you know things like fibrinogen which is involved in blood coagulation to cross into the brain and again as you mentioned the inflammation it's activating microglia cells and it's causing them
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
there is a fantastic researcher uh Katarina kasudu she is a Gladstone University UCS set value where she I think she developed an antibody that blocks the interaction between fibrinogen and microvia to avoid that over uh like over expression of information or over activation of microbial cells because she's also a strong believer that let's give it this functional very early and the major contributor to Dimension
now fibrinogen can activate uh the brain resident immune cells that are microbial cells through cd11b so that's a specific receptor so when fibrinogen gets in it can bind to microglia so it will induce an over reaction over inflammation of the brain which will be detrimental for uh for many things for the functions of the cell surrounding