The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
the HDL which let's say it's this other marker here they have another a different marker called an APO A1 they can have from one to five of them on their structure and it's not very accurate to measure that because what if one has one and one has five they're mostly in the form of Threes but generally they can have up to five on each HDL so this is very easy to measure when you measure an apob you measure like an exact particle
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the other family of lipoproteins are our high density lipoproteins are hdls their structural protein is APO protein A1 except there's from one to five copies of APO A1 per HDL particle so that doesn't become a useful biomarker to get an HDL particle concentration and the hdls until you get into the uh subd discussions of them per se are not atherogenic