The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
in the case of lpa it's we're you know we're very very confident that that we're using the the correct uh genetic instruments to infer a causal relationship between lpa and a wide range of uh atrocious cardiovascular diseases
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
lp little a is playing a causal role in the development of atherosclerotic cardiovascular disease independent of ldl
the effect of the variance on the outcome so the effect of lpa uh variants on cardiovascular disease are explained by higher lpa levels