The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the first is a molecule called A lipoprotein which Carri cholesterol through the body and specifically A lipoprotein that has an apob protein on it because there are lipoproteins that don't have apobs on them and we don't have to worry about those but the lipoproteins that have apobs on them can enter the artery wall when the endothelium uh is intact but they do so more prevalently and more easily when the endothelium is damaged
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's basically dependent on the number of particles in your bloodstream with apo B that are under 70 nmters because any lipoprotein under 70 nmters in diameter with an apo can penetrate the endothelium and get retained there.