The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I think what you said is true and I think it's just a that's more of a fact of just everybody across the board has marginal riboflavin status and if you have factors and increased riboflavin needs most people regardless of their diet are probably not getting enough yeah and not getting enough riboflavin can impair the function of your MTHFR lead to elevated levels of homocysteine and affect the biochemistry so
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
their homocysteine is too high and by too high it goes above 7 and that's not the only metric of riboflavin status but I see that repeatedly clinically
i riboflavin is like my favorite b vitamin because you know all methylation works with that mthfr has allosteric control with riboflavin apparently and so i see so often when people aren't getting their organs uh or getting you know riboflavin rich foods they don't methylate well their homocysteine goes up etc