Peter Attia· MD
when AN apob particle enters the aring wall it has a high Affinity to bind to Collective tissue molecules called proteoglycans sytin there's a whole sub family of them and now you have that apob particle that is just stuck there
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when AN apob particle enters the aring wall it has a high Affinity to bind to Collective tissue molecules called proteoglycans sytin there's a whole sub family of them and now you have that apob particle that is just stuck there
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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