Peter Attia· MD
i think for lpa it's it's a fairly uh fair assumption to make
Loading…
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
i think for lpa it's it's a fairly uh fair assumption to make
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's been shown that i probably the best study that has investigated that is uh is a study from the decode cohort which is a cohort from uh from iceland they have uh they sequenced uh they did a whole genome sequencing and i think it was fifty thousand ah sorry fifteen thousand uh individuals and they've shown unequivocally that the lpa isoform size even though you can sequence it really was not associated with the risk of heart attacks and strokes once you take into consideration lpa level so it's really the lpa number uh that matters
and that the isoform size matters because it's associated with different levels of of lpa and not through an independent effect
which is it mirrors apob the first second and third order factor in the fa in the uh then the harm caused by an ldl particle is the number of the particles the size all of those things only factor into the number