Paul Saladino· MD
so we should not use postprandial flow media Tillet ation as any sort of a proxy or metric for cardiovascular disease risk in general
We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
so we should not use postprandial flow media Tillet ation as any sort of a proxy or metric for cardiovascular disease risk in general
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
I do not think that any decline in flow mediated dilatation following food is a metric for is any its predictive in any way of of heart disease risk like that's a very poor indicator
my position on this is that flow mediated dilatation is a very poor predictor it's a very poor metric metric for cardiovascular disease risk