The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it's not that the arteries of the heart are uniquely susceptible to this process you just described as atherosclerosis it's just that two things uh are conspiring against us the first is that they are very small arteries and therefore it does not take a significant amount of obstruction or occlusion to create aeia which is just the technical term for when oxygen is no longer able to profuse the tissue
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's just that two things uh are conspiring against us the first is that they are very small arteries and therefore it does not take a significant amount of obstruction or occlusion to create esea which is just the technical term for when oxygen is no longer able to per use the tissue and then of course at the risk of stating the obvious the second fundamental problem is it happens to afflict an artery that is let's call it specifically sensitive to the demands of oxygen