The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
what happens is you're improving the flexibility of your arteries, you're making them more resilient. They're able to handle stress better. You're improving their overall functioning and you're making them more resilient and resistant to atheroscerosis, right?
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
And so the stronger the exercise, the more vigorous the exercise, the faster your heart is pumping. And that's why heart rate does come into this. The the more your blood flow is moving, right? So you're getting a stronger sheer stress. And that is actually not a bad type of stress. It's a good type of stress. The sheer stress causes your endothelial cells lining your arteries to adapt um and in a way where it improves vascular function. It improves endothelial function and it causes these endothelial cells to secrete beneficial molecules. So one of those would be nitric oxide. Many people have heard of that. And the other one would be prosttocyc.