The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
these LDL particles can essentially get stuck in circulation where they get smaller the odds of them becoming oxidized increases or modified by sugar you know glycated for example and they become pro-inflammatory so they become smaller they are more easily able to lodge themselves in the endothelium macrophages check them out there modify their in front pro-inflammatory they adhere to the you know endothelial wall and that's where you start to develop a foam cell
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
the size of the LDL particle really actually doesn't matter that much the size of the LDL particle can be a surrogate marker for insulin sensitivity