The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
we have different variants of it and so a subset of the population and it varies considerably by ethnicity so african uh east asian also quite high down to caucasian and uh and and um as you mentioned chinese japanese etc so you're going to see different expressions of the apo lipoprotein little a
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but we can say like in the ballpark that about 20 of the world population has an lpa level that puts them in a higher risk category so depending on uh ethnicity actually it can be very high as for instance in individuals of african ancestry who have the highest level and all the way down to uh chinese and japanese that probably have the lowest levels
but we can say like in the ballpark that uh about 20% of the world population has an LPA level that puts them in a higher risk uh category
now the good news perhaps is uh 80% of people don't inherit the gene that makes you produce the sapo protein little a but 20% do now people oh 20% no big deal look at 20% of the world population you're talking billions of people who have what's now recognized to be the number one uh lipid lipoprotein disorder associated with atherosclerosis