Paul Saladino· MD
if you the counter hypothesis is actually LDL is part of the recruitment for dealing with the disease particularly endothelial damage and dysfunction for which there actually is quite a lot of evidence already in the literature that suggests this for example the two easiest ones to mention improbable and largest ones you you'd mentioned earlier lipopolysaccharides that's in the wake of injecting and I think this happens with animal models and actually they may have done this with human models where they had a certain amount of life of polysaccharides injected into the blood the the liver up regulates low-density lipoproteins secretion and may even be direct with liberal protein secretion Logans elaborate protein secretion but regardless it's bringing these out to potentially fight the disease a lot of people don't know this but april-b containing lipoproteins also carry onboard alpha tocopherol which we tend to know as vitamin E which is in fact something that can connect with free radicals in our bloodstream reactive oxygen species and turn them into nonreactive products it's an antioxidant and it's carried on board low-density lipoproteins