The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it is going to be difficult to have some perturbation in a system that will lower or raise LDL that won't have some other effect the question is how do we with some reasonable degree of certainty look at those other effects and ask whether or not they're germane to the question of atherosclerosis and the causality of LDL to atherosclerosis
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.