Peter Attia· MD
I would love to see a clinical assay for a possi 3 & 4 LDL triglyceride concentration which goes back to a point we had a few minutes ago
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I would love to see a clinical assay for a possi 3 & 4 LDL triglyceride concentration which goes back to a point we had a few minutes ago
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
if you could add one clinical assay to the Arsenal of lipid ologists it would be an a possi three assay is that still true
so when you are presuming we're both insulin sensitive our VLDL really don't pose much of an Astra genic risk because they stick around for such a short period of time so we don't have many of them they don't stick around long our a possi 3 is quite down regulated but if a possi 3 is up regulated and it's hard to measure this although Sam tameka's is working on an assay to do so you have VLDL now start to act like LDL they stick around long enough they have a high enough residence time in the plasma that they become atherogenic