The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it's also transporting and this is where I was so intrigued inflammatory pain so cyto kindes caed protein also are being transported through vldl now that was important pre antibiotics pre everything that we do now to combat you know infectious disease and viruses and bacteria parasites whatever
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
But perhaps, even as importantly, it may serve an important role in host defense. And so I'll come to the LPS question at the end of this little discussion because a really interesting sort of corollary of that is, what role do lipoproteins have in the host defense mechanism?
But why would it need to come out in a hurry? Well, one of the arguments is it may be because it contains components, which it does, that help promote inflammation in the circulatory system. Why would that be important? Well, because when you have an organism, that'd be the parasite or another infectious agent in the vascular system, malaria or parasites, trypanosomas, whatever you care to mention, viruses, one way the body has of eliminating them is by setting up an inflammatory response and an immune response related to that. And VLDL carry pro-inflammatory proteins, they carry also pro-thrombotic proteins. They actually are a very efficient delivery system, sort of like a fire truck that carries a lot of things that we can use to fight a lot of infection.