The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
if you look at postprandial again so after a meal levels of TMAO the foods that stimulate the highest levels of TMAO are seafood and if you look at any study any any any type of study you choose to pick obviously most is going to be epidemiological but there are also some interventions seafood is associated with a reduced risk of cardiovascular disease
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basically what they're saying is tmao isn't bad for humans anyone that says that doesn't know the literature
indeed tmao has previously been suggested as a marker of seafood consumption tmao is much higher in seafood seafood is an important dietary source of free tmao in addition to tmao precursors carnitine and choline so fish has never been associated with cardiovascular disease
There's more preformed TMAO in many fish than the amount of TMAO you would make from the amount of choline and carnitine present in an equivalent amount of meat. But, fish is never really associated with cardiovascular disease, even in these observational studies.