The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
being able to have more particles that can scavenge more of these oxidized phospholipids and oxidized moieties and take them back to the liver which is the ultimate place of clearance for the LP little-a which is a totally safe place to take these things that would pose an advantage
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
more and more one of the functions perhaps even of apoa why it even evolved is a little scavenger protein that attaches to oxidized lipid moieties specifically oxidized phospholipids oxidized sterols they bind to it with great affinity and maybe that little garbage truck full of oxidized particles if it could bring it back to the liver or some other tissue that could catabolize it those oxidized lipid moieties which tend to be destructive to cells are not getting to cells
so we know that uh apoa can still have oxidized phospholipids