The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
endothelial cells actually have specific locks one receptors which is receptors for oxidized LDL the other thing called a sleeper receptor so to damage them in wonky LDL is very rapidly taken out of the circulation not only money into Celia cells but also in the liver
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
if you get a an ldl particle highly oxidized then understandably it's going to be removed from circulation by a scavenger receptor for example our endothelial cells have themselves scavenger receptors such as you know cd36 lox1 sortillin and so on and so forth they have binding domains specifically to grab modified ldl particles
then it is taken up by this locks one this uh oxidized LDL receptor which primarily um this has looked like it's occurring on the endothelial cells but it also can occur on macraga so we've got locks one receptors on endothelial cells and locks one receptors on macrofagos