Peter Attia· MD
I’m referring to all cells with LDLr that utilize receptor mediated endocytosis.
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I’m referring to all cells with LDLr that utilize receptor mediated endocytosis.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
no cells will make it or they'll get it by some free diffusion if they really need it or absolutely virtually any cell of it absolutely needed cholesterol because for some reason the synthesis was broken any cell could ultimately up regulate it held the L receptor but most of them don't so because they have no need for the cholesterol that's in an LDL the liver up regulates most your LDL receptors because that's involved clearance of these particles