The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
the lipid hypothesis is quite clear it's dose response it's dose dependent the higher the concentration the more atherosclerosis and so with high resolution ctm we should absolutely be able to identify existing plaques and observe the volume change
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
based on the lipid hypothesis, we would expect to see a linear effect of lifelong exposure to LDL on the risk of cardiovascular disease and mortality.