The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
only when there are a large number of oxidized LDL that the maccrate scavenger system the locks receptors are going to get overwhelmed and cause vascular disease so what you should look out here for is to find out what is causing the inflammation the LDL is there to protect us not to cause injury by itself it is a fireman it's not an arsonist
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
an injured LDL there is immediate signals that the body gives out to downregulate the ldlr receptor so the receptor that's picking up the normal LDL goes down and the locks receptor lox these are receptors that are present in the macro fages now these macr fages are there just like an army is not going to leave behind its injured army men the macres are going to go and pick up the oxidized LDL so that they are modulating and improving our response to injury
and only when there are a large number of oxidized LDL that the maccrate scavenger system the locks receptors are going to get overwhelmed and cause vascular disease so what you should look out here for is to find out what is causing the inflammation the LDL is there to protect us not to cause injury by itself