The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
basically means that you're trafficking more of these fatty acids for fuel and if you are you're trafficking them in something called a VLDL a very low density lipoprotein which is a precursor to LDL and therefore it makes sense why if you're being powered more by vldls or more specifically the cargo of triglycerides on vldls you'll end up with more LDLs
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
and in order for you to circulate those fatty acids at a faster and faster rate globally throughout your body it needs to resupply both those adipocytes and also to other oxidative tissues in need at a higher rate and it's doing that by packing ing them onto APO lipoproteins but particularly vldl when we're talking about the liver vldl is the um ultimately is succeeded by an LDL after lipoprotein lipase which is turning it over at a faster rate releases more of those fatty acids both to replete the diyes and also to power oxidative tissue like um especially skeletal muscle and especially cardiac uh myocytes