Paul Saladino· MD
the experiment intervention that they're talking about is flow media dilatation which is not the best measure for really atherosclerosis heart disease risk during these things
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We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
the experiment intervention that they're talking about is flow media dilatation which is not the best measure for really atherosclerosis heart disease risk during these things
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flow media dilatation is an extremely poor proxy for cardiovascular disease risk it's kind of like postprandial inflammation a lot of foods can increase post cranial inflammation a lot of foods are gonna change flow media dilatation and arteries this is not a good measure of endothelial function nor is it a good proxy of cardiovascular disease risk in any way shape or form right
it's not a good measure of cardiovascular disease risk