The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
and the reason for that is quite simple because when you measure ldl you also measure lpa cholesterol so if you have a very high ldl and also a very high lpa there's a very good chance that the high ldl cholesterol will actually be high lpa uh cholesterol
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
and variation in the lpa gene might even be the second cause of familial hypercholesterolemia
because when you measure ldl you also measure lpa cholesterol so if you have a very high ldl and also a very high lpa there's a very good chance that the high ldl cholesterol will actually be high lpa cholesterol