Lipoproteins like VLDL and LDL may bind to LPS as a protective mechanism to sequester it.

“as inflammation goes up, VLDL production is increased. And, at least in a couple of studies that I've read in literature, it seems to be implied the reason for that is that inflammation increases the release of endotoxin, which is a lipopolysaccharide from, you know, bacterial cell walls in the gut that gets released, and it binds to lipoproteins, to all lipoproteins, and it's sort of like an adaptive response to make sure we don't get sepsis or some sort of a toxic infection.”