The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the objective metric as with all patients is history first and very often in these families there is a family history of premature coronary disease that's one of the first things that speaks for these families then second these people have elevated LDL cholesterol without any other abnormality so hdl's normal triglycerides is normal LDL is elevated and you need to find elevated LDL in first degree relatives also so in Children of that individual or in one parent and in siblings
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
but unfortunately as you undoubtedly know there's overlap between no effect family members and affected family members in LDL cholesterol that's why we decided a long time ago to go for the mutation so to go for the genotype because for of course for the genotype there is no overlap either you carry the mutation or you don't carry the mutation