The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
a possi 3 is clearly the worst actor you could have here there's nothing worse than having an a possi 3 sitting on your a poby 100 cell in fact some would argue that may be the single worst thing you could ever have happen because it increases the residence time of them
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
what apoe and apoc3 do is that they regulate the speed of which things are happening they're different than apo b because they you can argue they're promiscuous apo b is matted to ldl particles maybe to some extent vldl ldl and this whole pipeline apoa1 to a large extent is matted to the hdl family of particles now apoe and apoc3 also known as exchangeable lipoproteins are not they can jump from different populations of lipoproteins and by taking different roles they can decide whether a lipoprotein is going to stay in the circulation and that would be your apoc3 or get cleared and that would be your apoe