The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I think what we're going to learn is that sort of that Glu Glu femoral storage capacity is going to be an important driver of risk in this in this context and finding ways to change that if it's possible would be of great benefit
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
but we think there are a group people here who repres present probably some polygenic version of this where there's a relative decrease in fat in the legs like I can think of patients now who come to my practice who may have a tiny little pot belly but you know their lipids were sort of not that bad and their legs are super skinny and they had a bypass at 38 or 40 or 41 or the number of people I can think of like that keeps getting bigger and bigger and I think what we're going to learn is that sort of that Glu Glu femoral storage capacity is going to be an important driver of risk in this in this context
but but but the question is this is probably also playing a very big role in people who aren't the ones showing up for byass at 38 years old who don't have the complete lipad distrophy where they have no ability to store uh fat on their on their legs and hips