Nutritional ketosis functions as a histone deacetylase inhibitor, rescuing the phenotype and circumventing the KMT 2d mutation in the Kabuki syndrome mouse model.

“nutritional ketosis functioning as a histone deacetylase inhibitor basically salvages or rescues the phenotype of this and circumvents this gene mutation the KMT 2d mutation the animals have a normal neuronal density in the dentate gyrus and even from a behavioral characteristic and enhances sort of learning and memory”