The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I think between the talks and evidence the fact that sporadic forms of Parkinson's disease can be associated with complex when deficiency or myocardial mutations I think helps to support the idea that mitochondrial dysfunction can play maybe in the causal path for Parkinson's disease
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
there are people who have Parkinson's and I apologize for my ignorance on this where they don't actually have a dopamine deficiency in the brain but they have a Parkinson's like phenotype purely from an insult to their mitochondria from say a toxin right so in these instances what we think has happened is that because of a environmental pesticide or insecticide the mitochondrion has been poisoned in some of these dopaminergic neurons and those neurons actually die so there is a dopamine loss