The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
So what’s interesting was they showed that if you block the inflammation right before the start of the stressor, sort of it starts, you block it, you can prevent the apoptosis, you can prevent the downstream behavioral effects, it’s protective.
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They showed that the stressor crazy activates inflammation, leads to apoptosis, death of microglial cells in the brain and, you know, huge anxious depressive behavior afterwards, right?
If you do nothing here and you let the little rodents go through the horrible stressor and you block the inflammation afterwards, they do worse. If you stimulate inflammation, they get antidepressant response.
if you block the inflammation right before the start of the stressors you sort of which it starts you block it you can prevent the apoptosis you can prevent the downstream behavioural effects its protective fine if you do nothing here and you let the little rodents go through the horrible stressor and you block inflammation afterwards they do worse if you stimulate inflammation they get an anti antidepressant response