The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
so if you if you have a slower smore sluggish empty Jabar gene you're not gonna be making that much methylfolate and you need that methyl Foley to take that homocysteine and slap a methyl group on it and now you're helping you make your methylation system again
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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so you know you homocysteine is good and you need almost cysteine you just methylate it and you methylated it with the methyl cobalamin in methyl foley which is what the energy bar gene makes
So taking, say, l-methylfolate could be a good option for me and I've experimented with that in the past, looking at how that can lower homocysteine or things like that.