The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
people who have gut dysfunction or gut inflammation may actually not be able to use short chain fatty acids as well from the gut and they may need to use butyrate from the bloodstream and the only way to do that is if you have ketones in your blood
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
if you look at the pathway even whether it's butyrate or isobutyrate coming into the clonic epithelial cell that's going to be transformed into beta hydroxybutyrate before it's used for fuel in those cells and you know becomes acetyl coa in the tca cycle