Peter Attia· MD
as we discussed earlier with CTP the directional flux of cholesterol in the blood blood is more from HDL to ape will be containing lipoproteins rather than the other way around so I'm pretty comfortable in saying I don't think that what you just outlined is a way that HDL AGL doesn't directly delipidate LDL no no I think it's more about if it's happening at all I want to say all this is couched in this is the Paradigm that we're dealing with but where there's still a lot of uncertainty about it but if it's happening at all it's really more that something about HDL or something apoa1 is more interacting with cells to promote efflux of cholesterol rather than with other lipoproteins like apob container hyper proteins