The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
if you have low luteinizing hormone which is the hormone produced by the pituitary gland that's generally showing that you have what would be considered secondary hypogonadism which means that the problem resides in your brain not with the testes
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
so if you have normal LH that means your body is telling your testes to make testosterone and if you have low testosterone in that case your testes aren't making it that would be primary hypogonadism
And throughout that whole cascade, you could have insufficient signal from that, insufficient response