The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it's when inflammation becomes more chronic um even after the acute problem has resolved or some times when it lingers that it becomes maladaptive and the balance tips against the organism or the host which is us so um you know a prolonged immune activation can lead to a persistent release of inflammatory cyto kindes or mediators and that can also damage healthy tissue
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
Would you do do you think that the hallmark of that is based on immune function i.e. deteriorated immune function andor overaggressive immune function or do you think no the hallmark of that could simply be found in a cytoine uh profile that is not typical?