The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
So in order for insulin to bind to its receptor, you have to have IRS1 and that that sort of regulates down downstream signaling from when insulin binds to its receptor. And sort of part of the molecular mechanism for insulin resistance quote unquote is sort of in inactivation of IRS1 which can be triggered by oxidative stress. So in states of diabetes, in states of obesity, we see excess reactive oxygen species.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.