The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
in some people who are glucose intolerant when they exceed their glucose threshold of intake or carbohydrate intake the mitochondria kind of flip out and you get more of these reactive oxygen species produced in the mitochondria and there goes the chain then you get this inflammatory type of insulin resistance it seems reasonable to me that this could be one of the mechanisms of an insulin resistant thing
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.