Paul Saladino· MD
insulin resistance could be driving the ability of LDL or the propensity of LDL to become retain in the sub and athelia later is a fascinating hypothesis
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
insulin resistance could be driving the ability of LDL or the propensity of LDL to become retain in the sub and athelia later is a fascinating hypothesis
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
so ldl is sending cholesterol to those arteries but that cholesterol is getting accumulated there because the potential because of a potential hold up in the repair process because of impairments in the immune system related to that hyperlyn selenia and metallogic dysfunction