The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the high blood glucose in the diabetic state is a function of impaired metabolism and upregulated gluconeogenesis and then upregulated fatty acid oxidation and the high insulin levels are because the insulin can't interact with the cell to have its effect to be internalized and degraded so it just floats around a lot much longer
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
the reason for the elevated insulin concentrations inside diabetics with so this is another thing they think oh high insulin levels are bad they have all these negative effects and this goes with the high blood glucose and everything because he talks about hypergluc insulinemia and Hyper um hypoglycemia the reason that you're getting hyper insulinemia is because when insulin docks on the receptor of the cell the cell internalizes insulin and degrades it but when the cells are resistant to insulin what winds up happening is you just get a lot of insulin floating around