The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
what they found was that when you gave a lot of fructose people got type 2 diabetes you could measure the oral glucose tolerance test and they actually became diabetic so what you see is that the fructose which is metabolized in liver is causing this fat accumulation specifically in the liver and that is going to cause hyperinsulinemia
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
because the liver is full of you know all this excess fat now you're trying to shove all this glucose into this fatty liver cell and the liver is like no I can't take anymore so I'm going to be resistant to this so as it gets resistant the blood glucose goes up and the body starts to have to make more insulin to get rid of this other situation which is I hey there's too much glucose in the body