The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
because of the different structure of the polyunsaturated fatty acids in beta oxidation and that changing ratio of fadh2 to nadh2 your fat cells become insulin sensitive when you eat polyunsaturated fatty acids it breaks your metabolism
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because of the way that beta oxidation of these polyunsaturated fatty acids happens it can affect the insulin sensitivity of the adipocyte
as a fatty acid is broken down in beta oxidation fadh2 and nadh2 are produced but the very important piece here is that when you break down polyunsaturated fatty acids or monounsaturated fatty acids they have a different ratio of fadh2 to nadh2
when you eat polyunsaturated fatty acids it breaks your metabolism
that excess polyunsaturates our excess double bonds in the in the molecules is going to really kind of muck up the ratio between fadh2 and any nadh in beta oxidation which is going to cause our cells to mean to remain sort of persistently insulin sensitive too long