The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
if your insulin resistant because there's liver fat or because of the cytokines coming in now you can't phosphorylate FOXO one you can transduce that insulin signal so now your glucose output goes up which then increases your serum glucose which then goes to your beta cell and acquires no off switch there's no off switch correct and your beta cell then has to make extra insulin and it's making it fasting it's not just making it in response to a meal it's making it all the time
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.