Rhonda Patrick· PhD
Damage that accumulates within cells can accelerate tie shortening and also causes cells including stem cells to become scinsesscent and non-functional.
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Damage that accumulates within cells can accelerate tie shortening and also causes cells including stem cells to become scinsesscent and non-functional.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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Do you agree that things that at least have been associated with DNA damage and telomere length in humans and associative studies may likely be the same things that help prevent the accumulation of cellular senescence?