Peter Attia· MD
So there's the the immune system in what in what model? Uh it's been done in two different models in mice. So this been done with the ERCC1 mutation. It's also been done by knocking down the the major trans tam the major transcription factor for mitochondria. So there's the the immune system in what in what model? Uh it's been done in two different models in mice. So this been done with the ERCC1 mutation. It's also been done by knocking down the the major trans tam the major transcription factor for mitochondria. So if you induce mitochondrial dysfunction only in the immune system, you induce secondary scinessence in the whole organ. Do you think that would be true in mammals? Sorry, in humans. It's that that's always it's a million-dollar question. in in some way the it's been shown in in two different models in mice B6 which which yeah B6 I don't remember the exact strain of the mouse but u uh there's no reason why it should be different frankly um and it speaks to the importance of the immune system the second way for the immune system is through chronic inflammation which is you know tied cause and effect in the whole aging process um now and we can talk about this later as well I find it fascinating you know the whole idea of chronic inflammation which is induced by the aging process but which itself actually further accelerates aging.